Our mission

Before the needle.

Get the critical information from the simplest, least painful sample there is, before a child is put through repeated needles, invasive procedures and long waits.
Axiisium  ·  a Vindicara company
The gap

An AML diagnosis is not finished until the genetics come back.

NPM1, FLT3, PML::RARA, the core-binding-factor fusions: they decide the treatment, the risk group, and whether a patient is eligible for a targeted drug. In most hospitals that panel takes three to ten days. In most of the world it never arrives.

While it runs, the patient waits, or treatment starts blind. And to get the sample that feeds it, a needle goes into the hip bone. For a child, under sedation. Then again at day fifteen to check response. Then again at the end of induction. Then again for residual disease.

Axiisium starts from a different fact. These mutations are not invisible. NPM1 mislocalises a nuclear protein and the nucleus folds into a cup. PML::RARA fills the cytoplasm with granules and Auer rods. The core-binding-factor fusions leave their own marks. Hematopathologists have suspected genotype from morphology for forty years. Axiisium is being built to do it systematically, on every cell of every case, and to state its own uncertainty gene by gene.

The mission, stated exactly

Predict molecular abnormalities from a routine blood smear, abstain when the visual evidence is insufficient, and progressively replace sequencing wherever prospective clinical trials prove it safe.

Three words, in order.

Predict

The model. Read the lesion off the cell, on Day 1, from the sample a blood draw already produces.

Abstain

The discipline that makes the model safe to use, because a system that guesses on the cases it cannot see is worse than no system.

Replace

The last word for a reason: it is earned in a prospective trial, one gene at a time, or it is not earned at all.

Why we exist

It started with a boy in the next bed.

As a child, our founder sat in a hospital ward beside a boy his own age with acute myeloid leukemia. It was Myanmar; the diagnosis was slow, and care was scarce. The boy never made it home.

That memory is the reason Axiisium exists. AML moves fast, and too often the answer cannot keep up. We are building the tool we wish that boy had had: one that reads what the cell already shows, in time to matter, and that can one day reach the kinds of places where the genetics never arrive at all.

Where we stand

Honest about the work.

Axiisium is early and in active development, a research-use tool today. We hold every claim to the same standard we hold the disease to, and we publish our results on real data as they stand, not before. Every number, with its conditions attached, is on the Evidence page.

What we can prove today →
Built for those facing acute myeloid leukemia

We are building this in time for the next child in that bed.

Orange is the leukemia awareness color.

Research Use Only. Not for diagnostic use.